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Par ailleurs, on constate une dГfaillance de lвOrganisation Mondiale de la SantГ OMS dans son soutien au gouvernement haГtien pour mettre en place un systГЁme de surveillance ГpidГmiologique efficace et rГactif. Or la qualitГ et la fiabilitГ des donnГes ГpidГmiologiques est essentielle pour optimiser la coordination des acteurs de secours et lвadaptation de la rГponse aux endroits les plus stratГgiques. In a preclinical ApoEвв mouse model, Stefan Roth and colleagues from the University of Munich demonstrated that stroke exacerbates atheroprogression through the propagation of the alarmin-mediated vascular fluspiral response.

The brain-released alarmin molecule high-mobility group protein 1, HMGB1 induces activation of monocytes and endothelium via receptor for advanced glycation end-products RAGE signaling cascade and through increased cell load. plaque and vulnerability. Furthermore, recruitment of activated monocytes through the CC chemokine receptor-ligand 2-CC chemokine receptor type 2 pathway was critical in stroke-induced vascular inflammation. Neutralization of circulating alarmins or knockdown of RAGE was also shown to attenuate atheroprogression. Finally, О-adrenoreceptor blockade decreased myeloid monocyte egress after stroke, while neutralization of circulating alarmins was necessary to reduce systemic loritex activation and aortic invasion.

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